Author

Andreas Wack

The Francis Crick InstituteORCID 0000-0001-5226-2991Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Organism:human· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

Stefania CrottaiD· 2 shared Adán Pinto-FernándeziD· 1 shared Alejandro Suárez‐BonnetiD· 1 shared Annemarthe G. van der VeeniD· 1 shared Antoni G. WrobeliD· 1 shared Benedikt Mathias KessleriD· 1 shared Calum C. BainiD· 1 shared Chrysante S. IliakisiD· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersImmunology

Stefania CrottaORCID

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3 papers

Recent·Most cited

ImmunologybioRxivPreprintMar 17, 2026

Diverse lung challenges elicit a conserved monocyte-to-macrophage differentiation blueprint

Chrysante S. Iliakis, Wouter T’Jonck, Isobel C. Mouat…·Calum C. Bain

The paper shows that monocyte-derived alveolar macrophages (mono-AMs) from different insults follow a conserved differentiation blueprint, while fetal-derived AMs show context-dependent changes.

DOI 10.64898/2026.03.15.711867

mousebulk RNA-seq

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ImmunologybioRxivPreprintMar 27, 2025

The ubiquitin E3 ligase Huwe1 facilitates viral and self RNA sensing by RIG-I-like receptors

Timo Oosenbrug, Dennis Gravekamp, Adán Pinto-Fernández…·Annemarthe G. van der Veen

This study shows Huwe1 is essential for RIG-I-like receptor-mediated type I and III IFN induction by ubiquitinating key pathway components like TRAF5.

DOI 10.1101/2025.03.27.645708

humanmouse

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GenomicsNature GeneticsPeer-reviewedOct 19, 2020

Tissue-specific and interferon-inducible expression of non-functional ACE2 through endogenous retrovirus co-option

Kevin W. Ng, Jan Attig, William Bolland…·George Kassiotis

The paper identifies a novel ACE2 isoform, LTR16A1-ACE2, induced by interferon, which is non-functional and does not enhance SARS-CoV-2 infection.

103 citations

humanbulk RNA-seq

𝕏 PostBluesky