Author

Melanie A MacMullan

Broad InstituteORCID 0000-0001-5296-5902Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Assay:proteomics· 2 Approach:statistical model· 2 Organism:human· 1 Assay:metabolomics· 1 Organism:mouse· 1 Organism:rat· 1

Frequent Co-Authors

Alireza DelfarahiD· 1 shared Andrea Guerrero· 1 shared Chi-Hong WuiD· 1 shared Dongqing Zheng· 1 shared Gina G. TurrigianoiD· 1 shared Hasmik KeshishianiD· 1 shared Jeffrey R. Cottrell· 1 shared Jonathan H. Sussman· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperSystems Biology

Alireza DelfarahORCID

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2 papers

Recent·Most cited

NeuroscienceeLifePeer-reviewedApr 26, 2022

A Bidirectional Switch in the Shank3 Phosphorylation State Biases Synapses toward Up or Down Scaling

Chi-Hong Wu, Vedakumar Tatavarty, Pierre MJ Beltran…·Gina G. Turrigiano

This study identifies bidirectional phosphorylation changes in Shank3 that regulate synaptic scaling up or down in mouse and rat neurons.

28 citations

Systems BiologyJournal of Cell SciencePeer-reviewedFeb 24, 2020

AKT but not MYC promotes reactive oxygen species-mediated cell death in oxidative culture

Dongqing Zheng, Jonathan H. Sussman, Matthew P. Jeon…·Nicholas Alexander Graham

The paper shows that AKT, but not MYC, causes cell death in oxidative conditions by increasing reactive oxygen species, revealing a metabolic vulnerability in cancer cells.

18 citations

humanproteomics