Author

William R. Scott

Imperial College Healthcare NHS TrustORCID 0000-0001-5467-114XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Physiology· 2 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

Liam McAllaniD· 2 shared AE TayloriD· 1 shared Akiko MammotoiD· 1 shared Amarah V. AnthonyiD· 1 shared Andrea WagenfeldiD· 1 shared Andrew D. Jenkinson· 1 shared Antonio M. A. MirandaiD· 1 shared Benjamin C. Lehne· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 15 mutual co-author links

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2 shared papersPhysiology

Liam McAllanORCID

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3 papers

Recent·Most cited

PhysiologybioRxivPreprintMar 30, 2026

Inhibition of the androgen-activating enzyme AKR1C3 selectively decreases systemic and intra-adipose 11-oxygenated androgens in women

Lina Schiffer, Amarah V. Anthony, Laura B. L. Wittemans…·Wiebke Arlt

Inhibition of AKR1C3 reduces 11-oxygenated androgens but not classic androgens in women.

DOI 10.64898/2026.03.27.714735

humanproteomics

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PhysiologyCirculation ResearchPeer-reviewedSep 5, 2024

EPAS1 Attenuates Atherosclerosis Initiation at Disturbed Flow Sites through Endothelial Fatty Acid Uptake

Daniela Pirri, Siyu Tian, Blanca Tardajos Ayllón…·Paul Charles Evans

This study shows EPAS1 reduces atherosclerosis at disturbed flow sites by promoting endothelial fatty acid uptake and metabolism, which is suppressed in obesity through triglyceride-PHD3 pathway.

23 citations

mouseproteomics

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Genetic & Genomic MedicineNature CommunicationsPeer-reviewedMay 15, 2023

Integrative genomic analyses in adipocytes implicate DNA methylation in human obesity and diabetes

Liam McAllan, Damir Baranas̆ić, Sergio Villicaña…·William R. Scott

This study identifies DNA methylation changes associated with extreme obesity and finds causal effects on obesity and metabolic disturbances using Mendelian Randomisation.

44 citationstop 4%

humanDNA methylation

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