Author

Jacqueline J. T. Marshall

The Francis Crick InstituteORCID 0000-0001-5491-2919Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biochemistry· 1 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2

Frequent Co-Authors

Angela ClerkiD· 2 shared Susanna T.E. CooperiD· 2 shared Alejandro Suárez‐BonnetiD· 1 shared Andrew C. CookiD· 1 shared Angus J.M. CameroniD· 1 shared Bernard M. SiowiD· 1 shared Christopher BarringtoniD· 1 shared Daniel J. StuckeyiD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersBiochemistry

Angela ClerkORCID

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2 papers

Recent·Most cited

BiochemistrybioRxivPreprintOct 28, 2026

PKNs are required for cardiac hypertrophy induced by angiotensin II in mice

Susanna T.E. Cooper, Jacqueline J. T. Marshall, Peter Parker, Angela Clerk

The paper investigates the role of PKN isoforms in cardiac hypertrophy induced by angiotensin II in mice, finding that Pkn1/3 are required for sustained hypertrophy while Pkn2 does not substitute for them.

DOI 10.64898/2026.10.26.761876

mouseexperimental assay

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Developmental BiologyBiochemical JournalPeer-reviewedJun 22, 2022

PKN2 deficiency leads both to prenatal congenital cardiomyopathy and defective angiotensin II stress responses.

Jacqueline J. T. Marshall, Joshua J. Cull, Hajed Obaid Alharbi…·Peter J. Parker

This study shows that PKN2 deficiency causes prenatal congenital cardiomyopathy and impairs angiotensin II-induced cardiac hypertrophy in adult mice.

9 citations

mouseexperimental assay

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