Anthony J. Baker

This study shows chronic treatment with a 1A-adrenergic agonist improves mitochondrial function and reverses right ventricular failure in a mouse model.

This paper develops a theoretical model to explain power-law stress relaxation and calcium-dependent passive mechanics in cardiac muscle by simulating domain unfolding in titin.

This study shows that Ca2+ increases cardiac muscle viscoelasticity independently of active force development, with a >6-fold increase in apparent viscoelasticity at higher Ca2+ levels.