Paolo Emanuele Forni

The paper uses lineage tracing to differentiate olfactory and vomeronasal neurons from GnRH-1 and terminal nerve neurons, revealing genetic heterogeneity and additional olfactory placode-derived cells in the brain.

This study shows that Cnpy1 is essential for V2R processing and transport in basal vomeronasal sensory neurons, with its loss causing functional impairment and circuit disruption.

The paper investigates the role of Yap and Taz in OEC and Schwann cell development using single-cell RNA sequencing and conditional ablation in mice, finding differential impacts without affecting GnRH-1 migration.

The paper introduces an inducible Cntn2CreERT2 mouse line to trace and manipulate early migratory neurons in the developing nose.

The paper identifies p63/Keratin5-positive basal cells in the vomeronasal organ's sensory and non-sensory epithelia, showing their role in self-renewal and differentiation into apical cells.

The paper investigates the role of Msl3 in meiotic entry in mice and finds that its loss-of-function does not disrupt spermatogenesis.

This study identifies Prokr2 expression in terminal nerve neurons and links their dysfunction to Kallmann syndrome through single-cell RNA sequencing and genetic tracing in mice.

The paper shows that AP-2ε reprograms vomeronasal sensory neurons and their behaviors by controlling gene expression, bypassing cellular plasticity restrictions.

This study identifies Notch1 receptor and Dll4 ligand differential expression in neuronal precursors and shows their role in determining the binary dichotomy of vomeronasal neuron types in mice.

This study identifies MSL3 as a master regulator coordinating transcription and translation during meiosis in female Drosophila and shows its conserved role in mouse spermatogenesis.

The paper develops a Python program to automate the quantification of glomeruli features in mouse olfactory bulbs, validated for efficiency and accuracy in genetic studies.

The paper identifies the genetic lineage of Isl1 in developing olfactory system cells and GnRH-1 neurons in mice using genetic and molecular techniques.

This study shows Gli3 is critical for olfactory ensheathing cell development and GnRH-1 neuronal migration, and identifies a GLI3 mutation in a Kallmann syndrome patient.

This study shows that Smad4 signaling is essential for the development, function, and circuit formation of basal vomeronasal sensory neurons.

This study shows that AP-2ε maintains basal vomeronasal sensory neuron identity by preventing apical gene expression and highlights their differentiation plasticity.

This study shows that proper olfactory bulb development and olfactory/vomeronasal axonal connections are not required for GnRH-1 neuronal migration to the hypothalamus.