David Voehringer

This study shows that persistent STAT6 activation in keratinocytes leads to chronic AD-like skin inflammation, pruritus, and S. aureus colonization in mice.

This study shows ILC2s regulate alveolar macrophage dynamics and function during secondary helminth infection by modulating their metabolism and parasite-killing capacity.

The paper identifies basophil recruitment to the spleen following TBI and shows their activation of B cells and dendritic cells via IL-13 signaling, enhancing protein translation through NORAD.

The paper shows that Il33-expressing cDC2s drive ILC2 expansion and eosinophilia in male mice during fungal airway inflammation, independent of ST2 and dependent on IL-4/13.

This study identifies IRF4 as a master regulator of multinucleated giant cell formation through integrated single-cell and bulk RNA sequencing, CRISPR/Cas9 editing, and in vivo fate mapping.

This study identifies AZIN2 as a regulator of adipocyte progenitor fate through polyamine metabolism, showing its protective role against obesity and metabolic dysfunction.

The paper identifies CSF1 and TGFβ as key signals for synovial lining macrophage development in mice, which occurs postnatally and is monocyte-independent.

This study shows that eosinophils contribute to chronic cutaneous leishmaniasis by expressing a unique metabolic profile that suppresses Th1 responses.

This study presents lineage tracing results from 18 myeloid-specific fate-mapping models in mouse peripheral blood and synovial tissue, validating synovial macrophage identification through flow cytometry and CITE-seq.

The paper identifies CD226high macrophages derived from MDP monocytes as a new myeloid cell type involved in lipid metabolism regulation across tissues.

The paper identifies IL-13 and its receptor as neuronal synaptic proteins that enhance synaptic activity and neuroprotection in response to injury.