Author

Haiyang Luo

University of MichiganORCID 0000-0001-6802-367XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:differential expression· 1 Organism:mouse· 1

Frequent Co-Authors

Chenyang Liu· 1 shared Han Liu· 1 shared Hang Zhang· 1 shared Henry Lauris PaulsoniD· 1 shared Honglin ZhengiD· 1 shared Kang Zhang· 1 shared Maria do Carmo CostaiD· 1 shared Na Zhang· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperNeuroscience

Chenyang Liu

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Seeded aggregation of ANXA11 induces prion-like propagation, TDP-43 co-pathology and nucleocytoplasmic transport defects

Haiyang Luo, Honglin Zheng, Yongting Lu, Chenyang Liu, Kang Zhang, Suying Duan, Hang Zhang, Yaochong Zhang, Y. Song, Tuo Wang, Han Liu, Na Zhang, Zongping Xia, Yuming Xu

This study shows ANXA11 forms prion-like amyloid fibrils through phase separation, which propagate between cells, induce TDP-43 pathology, and disrupt nucleocytoplasmic transport, linking ANXA11 dysfunction to ALS/FTD.

bioRxiv · Mar 26, 2026 · PreprintNeuroscience

Regional and age-dependent changes in ubiquitination in cellular and mouse models of Spinocerebellar ataxia type 3

Haiyang Luo, Sokol V. Todi, Henry Lauris Paulson, Maria do Carmo Costa

The paper investigates how wild-type and pathogenic ATXN3 affect ubiquitination levels in different brain regions and ages in SCA3 models.

Frontiers in Molecular Neuroscience · Apr 14, 2023Peer-reviewedNeuroscience