Tehila Atlan

The paper identifies molecular mechanisms that integrate environmental cues to trigger diapause exit in killifish embryos using transcriptomics and chemical screening.

This study models DDR syndromes in killifish and shows that cGAS-STING pathway activation contributes to disease phenotypes, with cGAS mutation partially rescuing defects.

This study shows that germline manipulation in killifish leads to sex-specific effects on longevity and repair mechanisms.

This study shows that mutating APRT in turquoise killifish extends male lifespan and improves metabolic health, with mitochondrial function and AMPK activity playing key roles.

This study identifies vgll3 as an antagonistically pleiotropic gene in turquoise killifish, showing it accelerates growth and reproduction but increases late-life disease risk and mortality.