Author

Marva Bergman

Hebrew University of JerusalemORCID 0000-0002-8970-7402Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Developmental Biology· 1 Genetics· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:Arabidopsis· 1 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:statistical model· 1 Organism:zebrafish· 1

Frequent Co-Authors

Itamar HareliD· 2 shared Tehila AtlaniD· 2 shared Aaron J.J. LemusiD· 1 shared Adi Oron-GottesmaniD· 1 shared Bérénice Anath BenayouniD· 1 shared Björn SchumacheriD· 1 shared David H. MeyeriD· 1 shared Eitan MosesiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersDevelopmental Biology

Itamar HarelORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

A molecular circuit integrates environmental cues to coordinate the exit from developmental dormancy

Omer Ben Dor, Itai Rozenberg, Tehila Atlan, Erin Davis, Marva Bergman, Adi Oron-Gottesman, Tommer Argaman, Walter Sandt, David H. Meyer, Yoav Rapoport, Mazal Tawil, Björn Schumacher, Jason E. Podrabsky, Michal Rabani, Itamar Harel

The paper identifies molecular mechanisms that integrate environmental cues to trigger diapause exit in killifish embryos using transcriptomics and chemical screening.

bioRxiv · Oct 27, 2026 · PreprintDevelopmental Biology

The cGAS-STING pathway is an in vivo modifier of genomic instability syndromes

Marva Bergman, Uri Goshtchevsky, Tehila Atlan, Gwendoline Astre, Ryan Halabi, Hosniyah El, Eitan Moses, Aaron J.J. Lemus, Bérénice Anath Benayoun, Yehuda Tzfati, Ido Ben‐Ami, Itamar Harel

This study models DDR syndromes in killifish and shows that cGAS-STING pathway activation contributes to disease phenotypes, with cGAS mutation partially rescuing defects.

bioRxiv · Oct 16, 2024 · PreprintGenetics