George A. Kassavetis

The paper shows that different binding modes to the nucleosome acidic patch lead to distinct cellular outcomes, with overexpression of Dot5 and camelid antibodies causing growth, DNA repair, and heterochromatin defects.

This study investigates the genome-wide transcriptional activities of HMGN proteins in human cells, revealing their role in activating gene expression at promoters and enhancers.

This study determines the structure of the Dsup and HMGN nucleosome-binding motif using cryo-EM, revealing their binding to the nucleosome acidic patch.