Author

Hella Luksch

Technische Universität DresdenORCID 0000-0001-7070-4992Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Approach:statistical model· 1

Frequent Co-Authors

Angela Rösen‐WolffiD· 2 shared Björn H. FalkenburgeriD· 2 shared Éva M. SzegőiD· 2 shared Andreas LinkermanniD· 1 shared Claudia GüntheriD· 1 shared David Geißler-LöschiD· 1 shared David Sprott· 1 shared Felix SchulzeiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersImmunology

Angela Rösen‐WolffORCID

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2 papers

Recent·Most cited

ImmunologyeLifePeer-reviewedMar 20, 2025

Tissue inflammation induced by constitutively active STING is mediated by enhanced TNF signaling

Hella Luksch, Felix Schulze, David Geißler-Lösch…·Angela Rösen‐Wolff

This study shows that constitutive STING activation in mice causes SAVI, and TNF signaling mediates its pathogenesis, with TNFR1/2 inhibition improving disease symptoms.

7 citations

mouseexperimental assay

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NeuroscienceeLifePeer-reviewedOct 31, 2022

Constitutively active STING causes neuroinflammation and degeneration of dopaminergic neurons in mice

Éva M. Szegő, Laura Malz, Nadine Rabe Bernhardt…·Hella Luksch

This study shows that constitutively active STING in mice leads to neuroinflammation and dopaminergic neuron degeneration, suggesting therapeutic targeting of this pathway.

94 citationstop 2%

mousestatistical model

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