Author

Angela Rösen‐Wolff

Klinik und Poliklinik für Kinder- und JugendmedizinORCID 0000-0002-9613-5879Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Approach:statistical model· 1

Frequent Co-Authors

Björn H. FalkenburgeriD· 2 shared Éva M. SzegőiD· 2 shared Hella LukschiD· 2 shared Andreas LinkermanniD· 1 shared Claudia GüntheriD· 1 shared David Geißler-LöschiD· 1 shared David Sprott· 1 shared Felix SchulzeiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersImmunology

Björn H. FalkenburgerORCID

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2 papers

Recent·Most cited

Tissue inflammation induced by constitutively active STING is mediated by enhanced TNF signaling

Hella Luksch, Felix Schulze, David Geißler-Lösch, David Sprott, Lennart Höfs, Éva M. Szegő, Wulf Tonnus, Stefan Winkler, Claudia Günther, Andreas Linkermann, Rayk Behrendt, Lino Lars Teichmann, Björn H. Falkenburger, Angela Rösen‐Wolff

This study shows that constitutive STING activation in mice causes SAVI, and TNF signaling mediates its pathogenesis, with TNFR1/2 inhibition improving disease symptoms.

eLife · Mar 20, 2025Peer-reviewedImmunology

Constitutively active STING causes neuroinflammation and degeneration of dopaminergic neurons in mice

Éva M. Szegő, Laura Malz, Nadine Rabe Bernhardt, Angela Rösen‐Wolff, Björn H. Falkenburger, Hella Luksch

This study shows that constitutively active STING in mice leads to neuroinflammation and dopaminergic neuron degeneration, suggesting therapeutic targeting of this pathway.

eLife · Oct 31, 2022Peer-reviewedNeuroscience