John G. Purdy

This study identifies glutamine as a carbon source for lipogenesis during HCMV infection through reductive carboxylation, contributing to lipidome remodeling.

This study shows HCMV increases phosphatidylcholine (PC) levels through de novo synthesis during early replication, independent of late gene expression.

This study shows HCMV infection reduces erucic acid levels, an antiviral fatty acid, by promoting metabolism that increases other fatty acids, thereby evading its antiviral effect.

This study shows that HCMV can replicate without glucose by using metabolites from upper glycolytic pathways like the pentose phosphate pathway.