Author

Masatake Tanaka

Kyushu UniversityORCID 0000-0001-7461-0678Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Organism:mouse· 1 Organism:rat· 1

Frequent Co-Authors

Daiki HorikamiiD· 1 shared Hui‐Chun HuangiD· 1 shared Jain Jeong· 1 shared James L. Boyer· 1 shared Koji ImotoiD· 1 shared Matthew J. McConnelliD· 1 shared Motoyuki KohjimaiD· 1 shared Nikolai Arefyev· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperPathology

Daiki HorikamiORCID

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2 papers

Recent·Most cited

Two types of regeneration mechanism in acute liver injury

Tomomi Aoyagi, Takeshi Goya, Koji Imoto, Yuki Azuma, Tomonobu Hioki, Motoyuki Kohjima, Masatake Tanaka, Yoshinao Oda, Yoshihiro Ogawa

This study identifies two distinct cell proliferation populations during liver regeneration in mice with acute liver injury using single-cell RNA-seq.

Stem Cell Reports · May 8, 2025Peer-reviewedPathology

Liver Lymphatic Dysfunction as a Driver of Fibrosis and Cirrhosis Progression

Jain Jeong, Shao‐Jung Hsu, Daiki Horikami, Teruo Utsumi, Yilin Yang, Nikolai Arefyev, Xuchen Zhang, Shi‐Ying Cai, James L. Boyer, Rolando Garcia-Milan, Masatake Tanaka, Matthew J. McConnell, Hui‐Chun Huang, Yasuko Iwakiri

This study shows that impaired liver lymphatic function drives fibrosis and cirrhosis progression and identifies VEGF-C as a potential therapeutic target.

bioRxiv · Jan 11, 2025 · PreprintPathology