Author

Iris Karina Gratz

University of SalzburgORCID 0000-0001-7470-7277Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Iris K Gratz1 paper · Johannes Kepler University Linz

Biology Disciplines

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Immunology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Approach:benchmark study· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1 Assay:flow cytometry· 1

Frequent Co-Authors

Daniel J. CampbelliD· 2 shared Suraj R. VarkhandeiD· 2 shared Aayush GuptaiD· 1 shared Angelika Stoecklinger· 1 shared Anshu Kumar SharmaiD· 1 shared Archana Kumari SinghiD· 1 shared Ariane Benedetti· 1 shared Daniel NeureiteriD· 1 shared

Activity by Year

2018-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersImmunology

Daniel J. CampbellORCID

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3 papers

Recent·Most cited

ImmunologyJournal of Investigative DermatologyPeer-reviewedApr 9, 2025

T cells promote distinct transcriptional programs of cutaneous inflammatory disease in keratinocytes and dermal fibroblasts

Hannah A. DeBerg, Mitchell L. Fahning, Suraj R. Varkhande…·Peter A. Morawski

The paper shows how different T cell types drive distinct transcriptional changes in skin cells during inflammation, linking these changes to diseases like psoriasis and scleroderma.

5 citations

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivPreprintJul 7, 2024

Hedgehog-Hippo pathway interactions promote T cell exclusion from the tumor microenvironment in basal cell carcinoma

Georg Stockmaier, Suraj R. Varkhande, Peter William Krenn…·Fritz Aberger

This study shows that HH/GLI and Hippo/YAP pathway interactions suppress T cell chemotaxis and promote immunosuppression in BCC by downregulating chemokine signaling.

DOI 10.1101/2024.07.07.602398

humanorganoid / primary culture

𝕏 PostBluesky

ImmunologybioRxivPreprintDec 9, 2018

Circulating CD4+ memory T cells give rise to a CD69+ resident memory T cell population in non-inflamed human skin

Maria M Klicznik, Ariane Benedetti, Angelika Stoecklinger…·Iris K Gratz

The paper shows circulating CD4+ memory T cells can generate CD4+CD69+ resident memory T cells in non-inflamed human skin without infection or inflammation.

DOI 10.1101/490094

humanflow cytometry

𝕏 PostBluesky