Christa Rhiner

This study shows that Drosophila can recover visual orientation behavior after optic lobe lesions, suggesting brain plasticity mechanisms are involved.

The paper identifies a bilateral communication module involving Fwe, Grindelwald/TNFR, and Veli/LIN-7 that mediates cell competition in Drosophila.

This study identifies extracellular proteoglycan remodeling as a shared mechanism driving brain repair and tumor progression through Hpsl and glypican interactions.

This study identifies Hbs and Rst adhesion molecules as regulators of cell elimination during epithelial remodelling in Drosophila, showing they define a regional code for cell removal.

The paper shows that removing less fit neurons in a Drosophila AD model reduces amyloid-β42-induced brain damage and cognitive/motor decline.