Author

Daniel Birnbaum

Centre National de la Recherche ScientifiqueORCID 0000-0001-7920-9883Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Daniel Birnbaum3 papers · Broad Institute

Biology Disciplines

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Cancer Biology· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Abdessamad El KaoutariiD· 1 shared Agathe CohendetiD· 1 shared Alexia LoprestiiD· 1 shared Anaïs AulasiD· 1 shared Anne MuratiiD· 1 shared Anthony GonçalvèsiD· 1 shared Arnaud GuilléiD· 1 shared Bernadette de RauglaudreiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCancer Biology

Abdessamad El KaoutariORCID

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2 papers

Recent·Most cited

Circulating colorectal tumor cells remodel their surfaceome to increase their viability and metastatic potential in the bloodstream

Olivier Cabaud, Anaïs Aulas, Alexia Lopresti, Claire Acquaviva, Pascal Finetti, Charlotte Dessaux, Laëtitia Ganier, Quentin Da Costa, Claire Germier, Lénaïg Mescam, Abdessamad El Kaoutari, Stéphane Audebert, Luc Camoin, Bernadette de Rauglaudre, Laurys Boudin, , Émilie Mamessier

The paper identifies PTK7 as a marker for colorectal cancer cell plasticity and metastatic potential, showing its loss in circulating tumor cells enables stress resistance and metastasis through surfaceome remodeling.

bioRxiv · Jul 27, 2026 · PreprintCancer Biology

Epigenenomic and transcriptomic characterization of NPM1-mutated CN-AML subtypes

Lia N’Guyen, Clara Tellez-Quijorna, Julien Vernerey, Sylvain Garciaz, Nadine Carbuccia, Arnaud Guillé, Claire Burny, Mathilde Poplineau, Anne Murati, Norbert Vey, Daniel Birnbaum, Estelle Duprez

The paper characterizes NPM1-mutated AML subtypes using epigenomic and transcriptomic data, revealing distinct clinical and molecular features based on H3K27me3 levels at the HIST1 cluster.

bioRxiv · May 7, 2024 · PreprintGenomics