Author

Huw W. Naylor

Cancer Research UK Cambridge CenterORCID 0000-0001-8264-8596Unclaimed ORCID profile · follow to get notified of new preprints

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Neuroscience· 1

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Research Focus:spatial domain detection· 1 Approach:statistical model· 1

Frequent Co-Authors

András LakatosiD· 1 shared Camilla LyonsiD· 1 shared Daniel LagosiD· 1 shared Denisa HathaziiD· 1 shared Éva MoravaiD· 1 shared İbrahim ElsharkawiiD· 1 shared Irena Josephina Johanna MuffelsiD· 1 shared Juliane S Müller· 1 shared

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2026-2026 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

András LakatosORCID

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1 paper

Recent·Most cited

Mitochondrial DNA heteroplasmy drives cortical neuronal disturbances in human organoids harbouring the common m.3243A>G mutation

Denisa Hathazi, Camilla Lyons, Daniel Lagos, Oliver Podmanicky, Mariana Zarate-Mendez, Yu Nie, Juliane S Müller, Kieren Simon James Allinson, Huw W. Naylor, Majlinda Lako, İbrahim Elsharkawi, Irena Josephina Johanna Muffels, Éva Morava, Tamás Kozicz, Patrick Francis Chinnery, , Rita Horváth

This study shows that high mitochondrial DNA heteroplasmy in human organoids with the m.3243A>G mutation causes cortical neuronal disturbances, including axonal degeneration and apoptosis, similar to MELAS patients.

DOAJ (DOAJ: Directory of Open Access Journals) · Jun 21, 2026Peer-reviewedNeuroscience