Hala Harony‐Nicolas

This study shows that Shank3-deficient rats have altered neural activity in the mesoaccumbens pathway, leading to impaired social reward processing, and that manipulating VTA activity can normalize behavior.

The paper investigates the role of glutamatergic neurons in the posterior intralaminar thalamus during social interactions, finding increased activity and specific behavioral effects.

This study identifies the supramammillary nucleus as a target of PVH oxytocin neurons and shows its necessity for social recognition memory in rats.

The paper identifies structural brain differences in male and female rat models of Fragile X syndrome, including reduced axonal caliber in the splenium of the corpus callosum.

This study shows that deleting the KH1 domain of Fmr1 in rats causes attention deficits and transcriptional changes in the medial prefrontal cortex, similar to FXS, and identifies potential therapeutic targets.