Author

Akira Yoshii

University of Illinois ChicagoORCID 0000-0001-8305-006XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:electronic health records· 1 Research Focus:spatial domain detection· 1

Frequent Co-Authors

Kevin P. KosteriD· 3 shared Zach FykeiD· 2 shared Adriana Caballero· 1 shared Amanda Niqula· 1 shared Anna I. ScottiD· 1 shared Daniel ChelskyiD· 1 shared Devin Wiley· 1 shared Edén Flores-BarreraiD· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersNeuroscience

Kevin P. KosterORCID

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3 papers

Recent·Most cited

NeuroscienceMolecular Genetics and MetabolismPeer-reviewedJul 15, 2024

Reduction of Neuroinflammation and Seizures in a Mouse Model of CLN1 Batten Disease using the Small Molecule Enzyme Mimetic, N-Tert-Butyl Hydroxylamine.

Zach Fyke, Rachel Johansson, Anna I. Scott…·Akira Yoshii

This study shows that NtBuHA reduces neuroinflammation and seizures in a Cln1-/- mouse model of CLN1 Batten Disease.

5 citations

mouseexperimental assay

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NeurosciencebioRxivPreprintDec 16, 2021

Loss of Depalmitoylation Exaggerates Synaptic Upscaling and Leads to Neuroinflammation in a Lysosomal Storage Disease

Kevin P. Koster, Edén Flores-Barrera, Emilce Artur de la Villarmois…·Akira Yoshii

This study shows that loss of PPT1, a depalmitoylating enzyme, leads to exaggerated synaptic upscaling and neuroinflammation in a lysosomal storage disease.

2 citations

mouseexperimental assay

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NeuroscienceeLifePeer-reviewedApr 4, 2019

NMDA Receptor Dysregulation by Defective Depalmitoylation in the Infantile Neuronal Ceroid Lipofuscinosis Mouse Model

Kevin P. Koster, Walter G. Francesconi, Fulvia Berton…·Akira Yoshii

This study shows that defective depalmitoylation in Ppt1-/- mice leads to NMDAR subunit stagnation, synaptic dysfunction, and increased excitotoxicity.

42 citations

mouseelectronic health records

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