Author

Maria Odenthal-Schnittler

University of MünsterORCID 0000-0001-8467-7585Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 2 Genomics· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Organism:human· 2 Assay:ATAC-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1

Frequent Co-Authors

Hans‐Joachim SchnittleriD· 4 shared Jan Philip KipckeiD· 2 shared Jochen Seebach· 2 shared Jonas FranziD· 2 shared Milos GaliciD· 2 shared Vesna Bojovic· 2 shared Ana VelićiD· 1 shared Arndt F. SiekmanniD· 1 shared

Activity by Year

2022-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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4 shared papersCell Biology

Hans‐Joachim SchnittlerORCID

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4 papers

Recent·Most cited

ImmunologyFrontiers in ImmunologyPeer-reviewedSep 9, 2025

TNF-a induces VE-cadherin-dependent gap/JAIL cycling through an intermediate state essential for neutrophil transmigration

Jan Philip Kipcke, Maria Odenthal-Schnittler, Mohammed Aldirawi…·Hans‐Joachim Schnittler

This study shows TNF-α induces an inflammatory morphological phenotype (IMP) through VE-cadherin-dependent gap/JAIL cycling, essential for neutrophil transmigration.

6 citationstop 5%

experimental assayhost-pathogen & immune response

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Cell BiologybioRxivPreprintAug 5, 2025

A critical role for VE-cadherin in regulating actin dynamics during endothelial maturation and non-inflammatory activation via a tension-sensitive intermediate state

Jonas Franz, Maria Odenthal-Schnittler, Jan Philip Kipcke…·Hans‐Joachim Schnittler

The paper identifies a tension-sensitive intermediate state in endothelial cells during non-inflammatory activation, which regulates actin dynamics and barrier function through VE-cadherin and MLC-dephosphorylation.

1 citation

humanbulk RNA-seq

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Cell BiologybioRxivPreprintJun 25, 2024

Mechanical Strain Activates Planar Cell Polarity Signaling to Coordinate Vascular Cell Dynamics

Lieke A. Golbach, Tanumoy Saha, Maria Odenthal-Schnittler…·Maja Matis

This study identifies Vangl1 as a key mediator of flow-dependent cell dynamics in vascular remodeling through planar cell polarity signaling.

1 citation

mouseimaging

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GenomicsScientific ReportsPeer-reviewedMar 21, 2022

DNA motif analysis of shear stress responsive endothelial enhancers reveals differential association of KLF and ETV/ETS binding sites with gained and lost enhancers

Roman Tsaryk, Nora Deniz Yucel, Elvin V. Leonard…·Arndt F. Siekmann

39 citations

humanzebrafish

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