Author

Jan Philip Kipcke

University of MünsterORCID 0009-0006-4526-0270Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

Hans‐Joachim SchnittleriD· 2 shared Jochen Seebach· 2 shared Jonas FranziD· 2 shared Maria Odenthal-SchnittleriD· 2 shared Vesna Bojovic· 2 shared Erez RaziD· 1 shared Franziska Merten· 1 shared Johannes Andreas EbleiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersImmunology

Hans‐Joachim SchnittlerORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

ImmunologyFrontiers in ImmunologyPeer-reviewedSep 9, 2025

TNF-a induces VE-cadherin-dependent gap/JAIL cycling through an intermediate state essential for neutrophil transmigration

Jan Philip Kipcke, Maria Odenthal-Schnittler, Mohammed Aldirawi…·Hans‐Joachim Schnittler

This study shows TNF-α induces an inflammatory morphological phenotype (IMP) through VE-cadherin-dependent gap/JAIL cycling, essential for neutrophil transmigration.

6 citationstop 5%

experimental assayhost-pathogen & immune response

𝕏 PostBluesky

Cell BiologybioRxivPreprintAug 5, 2025

A critical role for VE-cadherin in regulating actin dynamics during endothelial maturation and non-inflammatory activation via a tension-sensitive intermediate state

Jonas Franz, Maria Odenthal-Schnittler, Jan Philip Kipcke…·Hans‐Joachim Schnittler

The paper identifies a tension-sensitive intermediate state in endothelial cells during non-inflammatory activation, which regulates actin dynamics and barrier function through VE-cadherin and MLC-dephosphorylation.

1 citation

humanbulk RNA-seq

𝕏 PostBluesky