Author

Hiroyuki Kitao

Kyushu UniversityORCID 0000-0001-8682-2889Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Eiji OkiiD· 2 shared Hiroaki OchiiwaiD· 2 shared Koji AndoiD· 2 shared Makoto IimoriiD· 2 shared Takeshi WakasaiD· 2 shared Yoshihiko MaeharaiD· 2 shared Yuki KataokaiD· 2 shared Kentaro NonakaiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

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2 shared papersCell Biology

Eiji OkiORCID

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2 papers

Recent·Most cited

Temporal regulation of G2 phase avoids therapy-induced senescence caused by DNA replication stress-inducing drugs and provides synergistic cytotoxicity

Kentaro Nonaka, Takeshi Wakasa, Hiroaki Ochiiwa, Yuki Kataoka, Koji Ando, Eiji Oki, Tomoharu Yoshizumi, Yoshihiko Maehara, Hiroyuki Kitao, Makoto Iimori

This study shows that shortening the G2 phase enhances cell death from DNA replication stress-inducing drugs, regardless of p53 status.

bioRxiv · May 9, 2026 · PreprintCell Biology

IAP antagonists potentiate TNFα-triggered apoptosis but selectively eliminate senescent tumor cells independently of TNFα

Hiroaki Ochiiwa, Takeshi Wakasa, Yuki Kataoka, Koji Ando, Eiji Oki, Yoshihiko Maehara, Makoto Iimori, Hiroyuki Kitao

This study shows IAP antagonists induce apoptosis in senescent tumor cells both with and without TNF, suggesting they can eliminate TIS cells and sensitize non-senescent cells through TNF.

bioRxiv · Feb 3, 2025 · PreprintCancer Biology