Author

Eiji Oki

Kyushu UniversityORCID 0000-0002-9763-9366Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Research Focus:benchmarking· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Hiroaki OchiiwaiD· 2 shared Hiroyuki KitaoiD· 2 shared Koji AndoiD· 2 shared Makoto IimoriiD· 2 shared Takeshi WakasaiD· 2 shared Yoshihiko MaeharaiD· 2 shared Yuki KataokaiD· 2 shared Ayako SuzukiiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCell Biology

Hiroaki OchiiwaORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

SPATNIC enables H&E-grounded cancer-cell identification across spatial and single-cell transcriptomic modalities in colorectal cancer

Shunsuke A. Sakai, Ryosuke Nomura, Masaki Ohira, Kyoko Kurihara, SungGi Chi, Yuki Matsubara, Yukiko Morinaga, Takeshi Kato, Naoko Takeda-Miyata, Motohiro Kojima, Shingo Sakashita, Hiromichi Ebi, Rui Yamaguchi, Eiji Oki, Takao Fujisawa, , Riu Yamashita

SPATNIC is a variational autoencoder that identifies cancer cells from spatial and single-cell transcriptomic data in colorectal cancer, outperforming existing tools and identifying a rare cancer stem cell population.

bioRxiv · Oct 22, 2026 · PreprintCancer Biology

Temporal regulation of G2 phase avoids therapy-induced senescence caused by DNA replication stress-inducing drugs and provides synergistic cytotoxicity

Kentaro Nonaka, Takeshi Wakasa, Hiroaki Ochiiwa, Yuki Kataoka, Koji Ando, Eiji Oki, Tomoharu Yoshizumi, Yoshihiko Maehara, Hiroyuki Kitao, Makoto Iimori

This study shows that shortening the G2 phase enhances cell death from DNA replication stress-inducing drugs, regardless of p53 status.

bioRxiv · May 9, 2026 · PreprintCell Biology

IAP antagonists potentiate TNFα-triggered apoptosis but selectively eliminate senescent tumor cells independently of TNFα

Hiroaki Ochiiwa, Takeshi Wakasa, Yuki Kataoka, Koji Ando, Eiji Oki, Yoshihiko Maehara, Makoto Iimori, Hiroyuki Kitao

This study shows IAP antagonists induce apoptosis in senescent tumor cells both with and without TNF, suggesting they can eliminate TIS cells and sensitize non-senescent cells through TNF.

bioRxiv · Feb 3, 2025 · PreprintCancer Biology