Michael Schindler

This study identifies Vpr-mediated TCF7 degradation as a mechanism by which HIV-1 reduces CD4+ T cell stemness and promotes effector-like states.

This paper identifies CD81 as an Ebola virus inhibitor that is suppressed by GP and VP40, with implications for NFκB signaling and antiviral strategies.

This study shows that non-infected CD4+ T cells enhance replication of primary HIV-1 in macrophages and promote formation of virus-containing compartments (VCCs).

This study identifies platelet-neutrophil aggregates as contributors to psoriasis skin pathology and disease severity.