Author

Theresa M. Kisko

Philipps University of MarburgORCID 0000-0001-9010-9568Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:database / resource· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1 Organism:rat· 1

Frequent Co-Authors

Ayse Özge SunguriD· 2 shared Carlotta GilardiiD· 2 shared Frederike SteiniD· 2 shared Fridolin GroßiD· 2 shared Gerhard M. SchrattiD· 2 shared Helena Caria Martins· 2 shared Markus WöhriD· 2 shared Rainer K.W. SchwartingiD· 2 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersNeuroscience

Ayse Özge SungurORCID

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2 papers

Recent·Most cited

Early life stress-induced miR-708-5p regulates mood disorder-associated behavioural phenotypes in mice and is a potential diagnostic biomarker for bipolar disorder

Carlotta Gilardi, Helena Caria Martins, Alessandra Lo Bianco, Silvia Bicker, Pierre‐Luc Germain, Fridolin Groß, Ayse Özge Sungur, Theresa M. Kisko, Frederike Stein, Susanne L. Meinert, Rainer K.W. Schwarting, Markus Wöhr, Udo Dannlowski, Tilo T. J. Kircher, Gerhard M. Schratt

The paper identifies miR-708-5p as a biomarker for bipolar disorder and shows its role in regulating mood disorder-associated behaviors through targeting Nnat.

bioRxiv · Mar 14, 2024 · PreprintNeuroscience

Bipolar-associated miR-499-5p controls neuroplasticity by downregulating the Cav1.2 L-type voltage gated calcium channel subunit CACNB2

Helena Caria Martins, Ayse Özge Sungur, Carlotta Gilardi, Michael Alexander Pelzl, Silvia Bicker, Fridolin Groß, Jochen Winterer, Theresa M. Kisko, Natalia Malikowska‐Racia, Braun M.D., Katharina Brosch, Igor Nenadić, Frederike Stein, Susanne L. Meinert, Rainer K.W. Schwarting, , Gerhard M. Schratt

This study identifies miR-499-5p as a regulator of neuroplasticity in bipolar disorder by targeting CACNB2, leading to impaired dendritogenesis and cognitive function.

bioRxiv · Jun 9, 2021 · PreprintNeuroscience