Philipp Homan

The paper identifies two reproducible reward imaging phenotypes linked to dopamine D2 receptor function and shows differential responses to antipsychotic drugs.

The paper shows that natural speech can reveal the organization of conceptual cognitive maps, with disorganized speech linked to cognitive disorganization.

The paper shows that cognitive disorganization impairs the ability to form cognitive maps from visual input but not from linguistic structure.

The paper identifies amacrine cells as enriched in schizophrenia genetic risk and links this to synaptic biology, while finding retinal immune cells associated with multiple sclerosis.

This study links genetic risk for schizophrenia with retinal thinning using UK Biobank data, highlighting neuroinflammatory pathways as a potential mechanism.

This study investigates how structural brain alterations in schizophrenia relate to network architecture, finding hub susceptibility and shared epicenters across disease stages and disorders.

This study examines structural brain heterogeneity in schizophrenia using multimodal MRI data from the ENIGMA dataset, finding higher frontotemporal heterogeneity and left parahippocampal homogeneity in patients.

A technology-enhanced relapse prevention program reduced hospitalization days by 4 days compared to usual care in schizophrenia patients.

The paper examines how striatal volume and functional connectivity predict weight gain in early-phase psychosis patients undergoing antipsychotic treatment.

This study demonstrates that affective flexibility can occur unconsciously, as physiological responses adapt to reversed threat contingencies without conscious awareness of the stimuli.