Author

Kah Ying Ng

University of HelsinkiORCID 0000-0001-9312-937XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Brendan James BattersbyiD· 2 shared Alex E. EkvikiD· 1 shared Andrew Phillip WestiD· 1 shared Anı Akpınar· 1 shared Arafath Kaja NajumudeeniD· 1 shared Christopher Benjamin JacksoniD· 1 shared Clemens Raths· 1 shared Eli Berzell· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersCell Biology

Brendan James BattersbyORCID

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintOct 13, 2025

Intrinsic errors in mitochondrial translation trigger a decline in cell fitness.

Güleycan Lutfullahoğlu Bal, Kah Ying Ng, Eli Berzell…·Brendan James Battersby

This study shows that errors in mitochondrial translation cause cellular stress and metabolic changes linked to disease and aging, independent of oxidative phosphorylation.

DOI 10.1101/2025.10.13.682189

humanproteomics

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Molecular BiologyHuman Molecular GeneticsPeer-reviewedOct 25, 2021

Translation of MT-ATP6 pathogenic variants reveals distinct regulatory consequencesfrom the co-translational quality control of mitochondrial protein synthesis.

Kah Ying Ng, Uwe Richter, Christopher Benjamin Jackson…·Brendan James Battersby

The paper investigates how pathogenic variants in MT-ATP6 affect mitochondrial protein synthesis quality control and reveals distinct regulatory consequences on mitochondrial gene expression.

11 citations

humanproteomics

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