Author

Melinda S. Yates

University of North Carolina at Chapel HillORCID 0000-0001-9450-5517Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cancer Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:mouse· 2 Organism:human· 1

Frequent Co-Authors

Adam Allen· 1 shared Bing LiiD· 1 shared Brenda MelendeziD· 1 shared Claudia R. OlivaiD· 1 shared Collene R. JeteriD· 1 shared Corinne E. GrigueriD· 1 shared David Kyle MeyerholziD· 1 shared Deena M ScovilleiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

Adam Allen

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintApr 23, 2025

Oncogene SETDB1s Dual Role in Endometrial Cancer: Driving Tumor Progression and Immune Escape

Kiarash Salari, Jiaqing Hao, Ryan Jilek…·Shujie Yang

This study identifies SETDB1's dual role in endometrial cancer by promoting tumor growth and enabling immune evasion through gene regulation and macrophage suppression.

DOI 10.1101/2025.04.23.650291

mousebulk RNA-seq

𝕏 PostBluesky

Cancer BiologyJCI InsightPeer-reviewedFeb 18, 2025

Mitochondrial defects and metabolic vulnerabilities in Lynch syndrome-associated MSH2-deficient endometrial cancer

Mikayla Borthwick Bowen, Brenda Melendez, Qian Zhang…·Melinda S. Yates

This study identifies mitochondrial dysfunction and metabolic reprogramming in MSH2-deficient endometrial cancer, revealing a novel pathway and potential therapeutic targets.

11 citationstop 2%

mousehuman

𝕏 PostBluesky