Author

Sami Şanlıdağ

Åbo Akademi UniversityORCID 0000-0001-9777-8115Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Cecilia SahlgreniD· 2 shared Tommaso RistoriiD· 2 shared Bruno LarrivéeiD· 1 shared Christopher S. CheniD· 1 shared Erika HookeriD· 1 shared Hesam Hoursan· 1 shared Irene M. AspalteriD· 1 shared Kalonji Ntumba· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersDevelopmental Biology

Cecilia SahlgrenORCID

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2 papers

Recent·Most cited

Developmental BiologyDevelopmental CellPeer-reviewedFeb 5, 2026

Bmp9 regulates Notch signaling and the temporal dynamics of angiogenesis via Lunatic Fringe

Tommaso Ristori, Raphaël Thuret, Erika Hooker…·Katie Bentley

This study shows Bmp9 upregulates Lunatic Fringe in endothelial cells, enhancing Notch signaling and regulating angiogenesis dynamics.

9 citations

mousebulk RNA-seq

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Developmental BiologybioRxivPreprintMay 5, 2025

Receptor stoichiometry predicts artery-typical vulnerability to altered Notch signaling during smooth muscle differentiation.

Sami Şanlıdağ, Noora Virtanen, Hesam Hoursan…·Cecilia Sahlgren

This study shows Notch2 drives canonical Notch signaling in smooth muscle differentiation, while Notch3 requires higher doses to activate transcription due to weaker RBPJκ interaction, with implications for artery-specific diseases.

DOI 10.1101/2025.05.05.652186

mousestatistical model

𝕏 PostBluesky