Chelsea S. Bahney

This paper develops a machine learning-based behavioral phenotyping approach to quantify functional recovery in mice with tibia fractures, finding that pin stabilization improves recovery and that recovery patterns vary over time.

This study shows that muscarinic agonists can restore non-healing salivary glands by reactivating endogenous regeneration and mitochondrial function through cholinergic-calcium signaling.

This study shows that Zmpste24-/- mice have delayed fracture healing with immune dysregulation and increased cellular senescence compared to wild-type mice.

This study shows that β-catenin signaling regulates chondrocyte-to-osteoblast transformation during fracture healing and identifies its role in stem cell generation for repair.