Andrew D. Ratanatharathorn

The paper investigates DNA methylation mediation between PTSD severity and cardiovascular disease risk, identifying six CpGs as potential epigenetic biomarkers.

This paper conducts a trans-ancestry GWAS of hot flashes, identifies TACR3 as a key genetic target, and finds genetic overlaps with psychiatric disorders.

The paper uses machine learning to identify factors predicting prospective risk of post-traumatic stress symptoms, finding that prior PTSS, loneliness, and a specific DNA methylation site are key predictors.

The paper performs a meta-analysis of DNA methylation data from military cohorts to identify CpG sites and regions associated with PTSD development.

The paper identifies and validates modifiable factors for depression prevention using UK Biobank data and Mendelian randomization.

This study identifies ten novel methylation loci associated with PTSD through a meta-analysis of 1,896 cases and controls across ten cohorts, highlighting the AHRR locus and its link to kynurenine levels.

This study uses genetic data to show that educational attainment causally reduces PTSD risk, with economic status mediating this relationship.

This study conducts the largest GWAS for PTSD, identifying 6 genome-wide significant loci in European and African ancestries and highlighting novel biological pathways.