Javier Huayta

This study shows that sodium arsenite requires additional mitochondrial or redox stress to cause dopaminergic neurotoxicity in C. elegans, but not fluoroacetic acid.

This study investigates the neurotoxic effects of a PAH mixture across three species, finding neurotoxicity occurs through a non-AhR mechanism.

This study shows that inhibiting mitochondrial complex III with pesticides causes dopaminergic neurotoxicity in C. elegans through redox stress, which can be mitigated by antioxidants.

This study evaluates the effects of developmental neurotoxicants on neuronal architecture and function in C. elegans, identifying morphological and behavioral changes consistent with mammalian DNT literature.

The paper shows that morphological changes in dopaminergic neurons of C. elegans correlate with functional impairments, as worms with dendrite degeneration paralyze more in dopamine solutions.

This study quantifies DAF-16 spatiotemporal activity in C. elegans under dietary restriction and finds it predicts lifespan extension with 78% accuracy.