Author

Aliyah S. Webster

Duke UniversityORCID 0009-0005-3212-9647Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Pharmacology & Toxicology· 2

Signature Assays, Organisms & Methods

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Organism:C. elegans· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Assay:metabolomics· 1

Frequent Co-Authors

Javier HuaytaiD· 2 shared Joel N. MeyeriD· 2 shared Abigail S. JoyceiD· 1 shared Caroline BeggsiD· 1 shared Jay JhaveriD· 1 shared Kerry EttingeriD· 1 shared Melissa ChernickiD· 1 shared Nishad JayasundaraiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersPharmacology & Toxicology

Javier HuaytaORCID

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2 papers

Recent·Most cited

Pharmacology & ToxicologybioRxivPreprintAug 21, 2026

Evidence from three taxonomically distinct species for a non-AhR mechanism of developmental neurotoxicity of an environmentally derived mixture of polycyclic aromatic hydrocarbons

Samantha Phelps, Melissa Chernick, Javier Huayta…·Nishad Jayasundara

This study investigates the neurotoxic effects of a PAH mixture across three species, finding neurotoxicity occurs through a non-AhR mechanism.

DOI 10.64898/2026.08.12.743995

zebrafishC. elegans

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Pharmacology & ToxicologyRedox BiologyPeer-reviewedAug 13, 2026

Inhibition of mitochondrial complex III causes selective dopaminergic neurotoxicity by redox stress in Caenorhabditis elegans

Javier Huayta, Aliyah S. Webster, Jay Jhaver, Joel N. Meyer

This study shows that inhibiting mitochondrial complex III with pesticides causes dopaminergic neurotoxicity in C. elegans through redox stress, which can be mitigated by antioxidants.

DOI 10.1016/j.redox.2026.104351

C. elegansexperimental assay

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