Author

Teh-Wei Wang

Institute of Medical Science, The University of TokyoORCID 0000-0001-9874-2392Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:scRNA-seq· 2 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Makoto NakanishiiD· 2 shared Akio MorinobuiD· 1 shared Haruka TsuchiyaiD· 1 shared Hirofumi ShodaiD· 1 shared Ikuo TakazawaiD· 1 shared Kazuhiko YamamotoiD· 1 shared Kazuyoshi IshigakiiD· 1 shared Keishi FujioiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersImmunology

Makoto NakanishiORCID

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2 papers

Recent·Most cited

ImmunologybioRxivPreprintJun 29, 2026

An APOC1+ inflammatory CAF-like state drives a senescent, treatment-resistant niche in rheumatoid arthritis

Risa Yoshihara, Sotaro Nakajima, Reo Yamazato…·Keishi Fujio

The paper identifies a treatment-resistant fibroblast state in RA synovitis that drives chronic inflammation through senescence-associated mechanisms and highlights therapeutic targets beyond cytokine inhibition.

DOI 10.64898/2026.04.17.718831

humanscRNA-seq

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Cell BiologybioRxivPreprintJul 22, 2025

Single-cell-based evidence for GLS1 inhibitor as a bona fide senolytic agent in vivo

Yuki T. Okamura, Teh-Wei Wang, Satoshi Kawakami…·Makoto Nakanishi

This study uses single-cell RNA sequencing to show that the GLS1 inhibitor BPTES eliminates inflammatory senescent cells in aged mice and reduces chronic inflammation.

1 citation

mousescRNA-seq

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