Author

Hirofumi Shoda

The University of TokyoORCID 0000-0002-1536-0303Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetic & Genomic Medicine· 1 Genetics· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Kazuhiko YamamotoiD· 3 shared Kazuyoshi IshigakiiD· 3 shared Keishi FujioiD· 3 shared Haruka TsuchiyaiD· 2 shared Mineto OtaiD· 2 shared Takahiro ItamiyaiD· 2 shared Tomohisa OkamuraiD· 2 shared Yasuo NagafuchiiD· 2 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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3 shared papersImmunology

Kazuhiko YamamotoORCID

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3 papers

Recent·Most cited

ImmunologybioRxivPreprintJun 29, 2026

An APOC1+ inflammatory CAF-like state drives a senescent, treatment-resistant niche in rheumatoid arthritis

Risa Yoshihara, Sotaro Nakajima, Reo Yamazato…·Keishi Fujio

The paper identifies a treatment-resistant fibroblast state in RA synovitis that drives chronic inflammation through senescence-associated mechanisms and highlights therapeutic targets beyond cytokine inhibition.

DOI 10.64898/2026.04.17.718831

humanscRNA-seq

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Genetic & Genomic MedicinemedRxivPreprintJan 12, 2022

Cell-type-specific transcriptome architecture underlying the establishment and exacerbation of systemic lupus erythematosus

Masahiro Nakano, Mineto Ota, Yusuke Takeshima…·Keishi Fujio

This study identifies cell-type-specific transcriptomic signatures associated with SLE disease establishment and exacerbation, highlighting their clinical relevance and potential limitations in capturing genetic risk factors.

DOI 10.1101/2022.01.12.22269137

humanbulk RNA-seq

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GeneticsbioRxivPreprintDec 30, 2021

Splicing QTL analysis focusing on coding sequences reveals pathogenicity of disease susceptibility loci.

Kensuke Yamaguchi, Kazuyoshi Ishigaki, Akari Suzuki…·Yuta Kochi

The paper analyzes splicing QTLs focusing on coding sequences to identify disease-causal variants associated with GWAS traits through novel integration and selection approaches.

DOI 10.1101/2021.12.30.474578

humanlong-read sequencing

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