Author

Oluwaseun Akinyele

University of PittsburghORCID 0000-0002-0067-8191Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Anushe Munir· 2 shared Ashley C. NwaforiD· 1 shared Dwi U Kemaladewi· 1 shared Dwi U. KemaladewiiD· 1 shared Hülya BayıriD· 1 shared Jackson R. FoleyiD· 1 shared Jerry Vockley· 1 shared John WangiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersNeuroscience

Anushe Munir

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2 papers

Recent·Most cited

LAMA2-related muscular dystrophy alters Schwann cell calcium homeostasis and increases peripheral nerve inflammation

John Wang, Mallika Khati, Molly Heron, Anushe Munir, Oluwaseun Akinyele, Marie A Johnson, Jerry Vockley, Udai B Pandey, Dwi U Kemaladewi

This study examines LAMA2 deficiency in mice, showing altered Schwann cell calcium homeostasis and increased nerve inflammation into adulthood.

bioRxiv · Nov 2, 2026 · PreprintNeuroscience

Impaired polyamine metabolism causes behavioral and neuroanatomical defects in a novel mouse model of Snyder-Robinson Syndrome

Oluwaseun Akinyele, Anushe Munir, Marie A. Johnson, Megan S. Perez, Yuan Gao, Jackson R. Foley, Ashley C. Nwafor, Yijen Lin Wu, Tracy Murray Stewart, Robert A. Casero, Hülya Bayır, Dwi U. Kemaladewi

This paper characterizes a mouse model of Snyder-Robinson Syndrome with a missense mutation in the Sms gene, revealing behavioral, neuroanatomical, and metabolic defects.

Disease Models & Mechanisms · Mar 11, 2024Peer-reviewedNeuroscience