Author

Megan S. Perez

University of Pittsburgh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Research Focus:benchmarking· 2 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Approach:statistical model· 2 Research Focus:database / resource· 1

Frequent Co-Authors

Kyle D. KetchesiniD· 2 shared Aaron K. JenkinsiD· 1 shared Andrew D’Agostino· 1 shared Anushe Munir· 1 shared Ashley C. NwaforiD· 1 shared Chelsea A. VadnieiD· 1 shared Colleen A. McClungiD· 1 shared Dwi U. KemaladewiiD· 1 shared

Activity by Year

2024-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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2 shared papersNeuroscience

Kyle D. KetchesinORCID

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Author profile →

3 papers

Recent·Most cited

Striatal output regulates the postnatal maturation of cortical circuits

Michael Janeček, Tara Deemyad, Yi-Chun Shih, Vicente Valle, Andrew D’Agostino, Michael Matarazzo, Megan S. Perez, Kyle D. Ketchesin, Susana da Silva, Rui T. Peixoto

This study shows that striatal output during early postnatal development affects the activity and maturation of the dorsomedial prefrontal cortex circuits in mice.

Cell Reports · Aug 23, 2025Peer-reviewedNeuroscience

Comparative transcriptomic rhythms in the mouse and human prefrontal cortex

Jennifer N. Burns, Aaron K. Jenkins, Xiangning Xue, Kaitlyn A. Petersen, Kyle D. Ketchesin, Megan S. Perez, Chelsea A. Vadnie, Madeline R. Scott, Marianne L. Seney, George C. Tseng, Colleen A. McClung

This study compares circadian transcriptomic rhythms in mouse and human prefrontal cortex subregions, finding conserved signaling with sex-specific phase shifts and species-specific rhythmic transcript identities.

Frontiers in Neuroscience · Jan 13, 2025Peer-reviewedNeuroscience

Impaired polyamine metabolism causes behavioral and neuroanatomical defects in a novel mouse model of Snyder-Robinson Syndrome

Oluwaseun Akinyele, Anushe Munir, Marie A. Johnson, Megan S. Perez, Yuan Gao, Jackson R. Foley, Ashley C. Nwafor, Yijen Lin Wu, Tracy Murray Stewart, Robert A. Casero, Hülya Bayır, Dwi U. Kemaladewi

This paper characterizes a mouse model of Snyder-Robinson Syndrome with a missense mutation in the Sms gene, revealing behavioral, neuroanatomical, and metabolic defects.

Disease Models & Mechanisms · Mar 11, 2024Peer-reviewedNeuroscience