Author

Anushe Munir

University of Pittsburgh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1 Organism:human· 1

Frequent Co-Authors

Dwi U. KemaladewiiD· 2 shared Marie A. Johnson· 2 shared Oluwaseun AkinyeleiD· 2 shared Annie I. Arockiaraj· 1 shared Ashley C. NwaforiD· 1 shared Dwi U Kemaladewi· 1 shared Hülya BayıriD· 1 shared Jackson R. FoleyiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersNeuroscience

Dwi U. KemaladewiORCID

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Author profile →

3 papers

Recent·Most cited

LAMA2-related muscular dystrophy alters Schwann cell calcium homeostasis and increases peripheral nerve inflammation

John Wang, Mallika Khati, Molly Heron, Anushe Munir, Oluwaseun Akinyele, Marie A Johnson, Jerry Vockley, Udai B Pandey, Dwi U Kemaladewi

This study examines LAMA2 deficiency in mice, showing altered Schwann cell calcium homeostasis and increased nerve inflammation into adulthood.

bioRxiv · Nov 2, 2026 · PreprintNeuroscience

Impaired polyamine metabolism causes behavioral and neuroanatomical defects in a novel mouse model of Snyder-Robinson Syndrome

Oluwaseun Akinyele, Anushe Munir, Marie A. Johnson, Megan S. Perez, Yuan Gao, Jackson R. Foley, Ashley C. Nwafor, Yijen Lin Wu, Tracy Murray Stewart, Robert A. Casero, Hülya Bayır, Dwi U. Kemaladewi

This paper characterizes a mouse model of Snyder-Robinson Syndrome with a missense mutation in the Sms gene, revealing behavioral, neuroanatomical, and metabolic defects.

Disease Models & Mechanisms · Mar 11, 2024Peer-reviewedNeuroscience

CRISPRa-induced upregulation of human LAMA1 compensates for LAMA2-deficiency in Merosin-deficient congenital muscular dystrophy

Annie I. Arockiaraj, Marie A. Johnson, Anushe Munir, Prasanna Ekambaram, Peter C. Lucas, Linda M. McAllister‐Lucas, Dwi U. Kemaladewi

This study shows that CRISPRa-mediated upregulation of LAMA1 compensates for LAMA2 deficiency in MDC1A fibroblasts, reducing cell migration and related pathway activity.

bioRxiv · Mar 6, 2023 · PreprintGenetics