Author

Ines Rosignol

German Center for Neurodegenerative DiseasesORCID 0000-0002-0185-2999Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Organism:zebrafish· 1

Frequent Co-Authors

Natalia Rodríguez‐MuelaiD· 2 shared Tobias GraßiD· 2 shared Zeynep DokuzluogluiD· 2 shared Ana‐Maria OprişoreanuiD· 1 shared Anna Dalinskaya· 1 shared Antonio Caldarelli· 1 shared Björn H. FalkenburgeriD· 1 shared Brunhilde WirthiD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersNeuroscience

Natalia Rodríguez‐MuelaORCID

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2 papers

Recent·Most cited

Altered lysosomal biology impairs motor neuron survival via TFEB dysregulation in spinal muscular atrophy

Ines Rosignol, Zeynep Dokuzluoglu, Antonio Caldarelli, Ana‐Maria Oprişoreanu, Sofiia Ushakova, Tohid Siddiqui, Rahul Grover, Hannes Guerlich, Björn H. Falkenburger, Stefan Diez, Tobias Graß, Catherina Gwynne Becker, Natalia Rodríguez‐Muela

The paper shows that TFEB activation restores lysosomal function and improves motor neuron survival in SMA, highlighting its potential as a therapeutic target.

bioRxiv · Jul 7, 2025 · PreprintNeuroscience

An isogenic human iPSC model unravels neurodevelopmental abnormalities in SMA

Tobias Graß, Ines Rosignol, Joshua D. Thomas, Felix Buchner, Zeynep Dokuzluoglu, Anna Dalinskaya, Jutta Becker, Brunhilde Wirth, Natalia Rodríguez‐Muela

This study uses an isogenic human iPSC model to show altered neural progenitor and motor neuron markers in SMA, suggesting developmental defects may contribute to neuronal death.

bioRxiv · Jan 2, 2023 · PreprintNeuroscience