Author

Loélia Babin

Centre National de la Recherche ScientifiqueORCID 0000-0002-0188-6582Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:statistical model· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:gene regulatory network inference· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Chloé BessièreiD· 2 shared Fabienne MeggettoiD· 2 shared Laurence LamantiD· 2 shared Sandra DailhauiD· 2 shared Stéphane PyronnetiD· 2 shared Ahmed Zamani· 1 shared Ayeong So· 1 shared Bernard S. LópeziD· 1 shared

Activity by Year

2019-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCancer Biology

Chloé BessièreORCID

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3 papers

Recent·Most cited

CircZBTB46, a promising therapeutic target in crizotinib resistant ALK-positive T lymphomas

Loélia Babin, Elissa Andraos, Steffen Fuchs, Chloé Bessière, Lola Colras, Sandra Dailhau, Cathy Quelen, Cindy Pinto, Iyad Daoudi, Romain Pfeifer, Ahmed Zamani, Marina Bousquet, Stéphane Pyronnet, Christine Gaspin, Laurence Lamant, Fabienne Meggetto

The paper identifies circZBTB46 as a driver of crizotinib resistance in ALK-positive lymphomas and shows its targeting restores drug sensitivity.

Leukemia · Jun 26, 2026Peer-reviewedCancer Biology

Tumor microenvironment signature associated with morphology in systemic ALK-positive anaplastic large cell lymphoma

Chloé Bessière, Loélia Babin, Sandra Dailhau, Philippe Gaulard, Stéphane Pyronnet, Fabienne Meggetto, Laurence Lamant

The paper identifies tumor microenvironment signatures linked to morphology and prognosis in ALK-positive anaplastic large cell lymphoma using computational analysis of gene expression data.

bioRxiv · Jan 20, 2026 · PreprintCancer Biology

RAD51 promotes non-homologous genetic rearrangements that are prevented by 53BP1

Josée Guirouilh‐Barbat, Wei Yu, Loélia Babin, Elisa Yaniz‐Galende, Tatiana Valentinovna Popova, Ayeong So, Chloé Lescale, Marc‐Henri Stern, Erika Brunet, Ludovic Deriano, Bernard S. López

The paper shows RAD51 and BRCA2 promote non-homologous genetic rearrangements, which are prevented by 53BP1, and links BRCA2 proficiency to increased non-homologous sequence capture in breast tumors.

bioRxiv · Sep 14, 2019 · PreprintMolecular Biology