Author

Raphael E. Pollock

The Ohio State University Wexner Medical CenterORCID 0000-0002-0443-1583Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Fernanda Costas Casal de FariaiD· 2 shared Valerie Patrice GrignoliD· 2 shared Abeba ZewduiD· 1 shared Ali F. UsmaniiD· 1 shared Connor P Harper· 1 shared Danielle BraggioiD· 1 shared David Koller· 1 shared David WangiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCancer Biology

Fernanda Costas Casal de FariaORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintAug 10, 2026

Extracellular vesicle-mediated suppression of macrophage STING signaling promotes immune dysfunction in dedifferentiated liposarcoma

Qi Zhang, Jay K. Mandula, Patricia Sarchet…·Federica Calore

This study shows that extracellular vesicles from dedifferentiated liposarcoma suppress macrophage STING signaling, leading to immune dysfunction and poor response to immunotherapy.

DOI 10.64898/2026.08.07.743624

humanbulk RNA-seq

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Molecular BiologyPLoS ONEPeer-reviewedSep 17, 2025

Blockade of Interleukin-6 (IL-6) Signaling in Dedifferentiated Liposarcoma (DDLPS) Decreases Mouse Double Minute 2 (MDM2) Oncogenicity via Alternative Splicing

Abeba Zewdu, Danielle Braggio, Gonzalo Lopez…·Raphael E. Pollock

This study shows that blocking IL-6 signaling in DDLPS reduces MDM2 oncogenicity through alternative splicing, suggesting a potential therapeutic target.

2 citations

mousebulk RNA-seq

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