Author

Lipeng Mao

ORCID 0000-0002-0629-0307Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Organism:human· 1 Approach:machine learning (classical)· 1 Organism:mouse· 1 Research Focus:protein design· 1 Research Focus:protein structure prediction· 1 Approach:statistical model· 1

Frequent Co-Authors

Beatriz Escobar-DonceliD· 1 shared Chenglong XieiD· 1 shared Evandro Fei FangiD· 1 shared Farrukh Abbas ChaudhryiD· 1 shared Guang YangiD· 1 shared Guobing CheniD· 1 shared Hidehito KuroyanagiiD· 1 shared Jon Storm‐MathiseniD· 1 shared

Activity by Year

2024-2024 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Beatriz Escobar-DoncelORCID

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1 paper

Recent·Most cited

NAD+ - and EVA1-C-dependent reversal of neurological deficits is mediated by differential alternative RNA splicing in tauopathic animal models

Ruixue Ai, Lipeng Mao, Xurui Jin, Shiqi Zhang, Junping Pan, Mariá José Lagartos-Donate, Shuqin Cao, Guang Yang, Chenglong Xie, Xiongbin Kang, Pingjie Wang, Yang Hu, Linda Hildegard Bergersen, Jon Storm‐Mathisen, Hidehito Kuroyanagi, , Evandro Fei Fang

The paper shows NAD+ and EVA1-C regulate alternative splicing in tauopathies, affecting protein interactions and disease progression, suggesting therapeutic potential for AD.

bioRxiv · Oct 27, 2024 · PreprintNeuroscience