Author

Jon Storm‐Mathisen

University of OsloORCID 0000-0002-2930-3262Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2 Animal Behavior & Cognition· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Organism:mouse· 3 Research Focus:differential expression· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Farrukh Abbas ChaudhryiD· 3 shared Linda Hildegard BergerseniD· 2 shared Beatriz Escobar-DonceliD· 1 shared Can Tartanoglu· 1 shared Chenglong XieiD· 1 shared Daxin WangiD· 1 shared Diana DomańskaiD· 1 shared Evan Michael VallenariiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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3 shared papersNeuroscience

Farrukh Abbas ChaudhryORCID

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3 papers

Recent·Most cited

Lactate treatment improves brain biochemistry and cognitive function in transgenic Alzheimer's and wild-type mice

Imen Belhaj, Ingrid Åmellem, Can Tartanoglu, Hanne M. Weidemann, Evan Michael Vallenari, Mingyi Yang, Farrukh Abbas Chaudhry, Magnar Bjørås, Jon Storm‐Mathisen, Linda Hildegard Bergersen

This study shows lactate treatment improves cognitive function and molecular biomarkers in Alzheimer's mice and wild-type controls.

Scientific Reports · Apr 14, 2026Peer-reviewedAnimal Behavior & Cognition

The glutamine transporter Slc38a1 is widely expressed in the embryonic neurogenic niches and impacts neuronal volume, survival, and morphology

Daxin Wang, Marivi Nabong Moen, Diana Domańska, Muhammad Zahoor, Wenjing Cai, Ragnhild E. Paulsen, Tor Paaske Utheim, Frode Lars Jahnsen, Jon Storm‐Mathisen, Farrukh Abbas Chaudhry

This study investigates the role of Slc38a1 in neurogenesis and brain development, finding it essential for embryonic neurodevelopment and impacting neuronal morphology and viability.

bioRxiv · Dec 18, 2025 · PreprintNeuroscience

NAD+ - and EVA1-C-dependent reversal of neurological deficits is mediated by differential alternative RNA splicing in tauopathic animal models

Ruixue Ai, Lipeng Mao, Xurui Jin, Shiqi Zhang, Junping Pan, Mariá José Lagartos-Donate, Shuqin Cao, Guang Yang, Chenglong Xie, Xiongbin Kang, Pingjie Wang, Yang Hu, Linda Hildegard Bergersen, Jon Storm‐Mathisen, Hidehito Kuroyanagi, , Evandro Fei Fang

The paper shows NAD+ and EVA1-C regulate alternative splicing in tauopathies, affecting protein interactions and disease progression, suggesting therapeutic potential for AD.

bioRxiv · Oct 27, 2024 · PreprintNeuroscience