Author

Hang Lin

University of PittsburghORCID 0000-0002-0781-6630Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Molecular Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Assay:imaging· 1

Frequent Co-Authors

Allison BeaniD· 1 shared Christopher H. EvansiD· 1 shared Fabrisia AmbrosioiD· 1 shared Gabrielle G. GilmeriD· 1 shared Hirotaka IijimaiD· 1 shared Jingying LiuiD· 1 shared Kai WangiD· 1 shared Lina Su· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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1 shared paperMolecular Biology

Allison BeanORCID

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3 papers

Recent·Most cited

PathologyScientific ReportsPeer-reviewedSep 2, 2026

Imaging-Based Age- and Sex-Related Cardiovascular and Skeletal Phenotypes in Fbn1C1041G/+ Mice

Wenxin Liu, Shuolei Li, Xiangyu Zhang…·Sufang Li

This study identifies age- and sex-dependent cardiovascular and skeletal changes in Fbn1C1041G/+ mice using imaging techniques.

DOI 10.1038/s41598-026-69281-0

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Cell BiologyOsteoarthritis and CartilagePeer-reviewedApr 1, 2025

Aging-associated Increase of GATA4 levels in Articular Cartilage is Linked to Impaired Regenerative Capacity of Chondrocytes and Osteoarthritis

Meagan J. Makarczyk, Yiqian Zhang, Olivia Bartholomew…·Hang Lin

This study identifies GATA4 as a key factor in chondrocyte aging and osteoarthritis, showing its role in impaired regeneration and inflammation.

1 citation

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Molecular BiologybioRxivPreprintMar 13, 2021

Age-related increase in matrix stiffness downregulates α-Klotho in chondrocytes and induces cartilage degeneration

Hirotaka Iijima, Gabrielle G. Gilmer, Kai Wang…·Fabrisia Ambrosio

This study identifies age-related matrix stiffness as a driver of decreased α-Klotho in chondrocytes, leading to cartilage degeneration in mice and humans.

1 citation

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