Atsushi Takata

The paper identifies NK cell activation as a genetic and functional contributor to MDD susceptibility using single-cell transcriptomics and population genetics.

This study uses reversal learning tasks and computational modeling to identify distinct cognitive impairments in mouse models of neurodevelopmental disorders.

This study identifies and characterizes genome-wide QTLs for transcriptional noise in human midbrain cells, revealing distinct patterns and potential disease associations.

This study shows that Setd1a-deficient mice exhibit schizophrenia-like phenotypes, which can be reversed by restoring Setd1a expression or inhibiting LSD1.