Author

Anastasia Diamantopoulou

Columbia UniversityORCID 0000-0002-3350-0330Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Bin XuiD· 2 shared Joseph A. GogosiD· 2 shared Pratibha ThakuriD· 2 shared Annie Ferng· 1 shared Atsushi TakataiD· 1 shared Chia‐Yuan ChangiD· 1 shared Curt MazuriD· 1 shared Enrico Cannavò· 1 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersNeuroscience

Bin XuORCID

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Author profile →

2 papers

Recent·Most cited

An antisense oligonucleotide-based strategy to ameliorate cognitive dysfunction in the 22q11.2 Deletion Syndrome

Pratibha Thakur, Martin Lackinger, Anastasia Diamantopoulou, Sneha B. Rao, Yijing Chen, Khakima Khalizova, Annie Ferng, Curt Mazur, Holly B. Kordasiewicz, Robert J. Shprintzen, Sander Markx, Bin Xu, Joseph A. Gogos

This study shows that reducing EMC10 levels in 22q11.2 deletion syndrome models improves cognitive function and sustains benefits for over two months.

eLife · Apr 17, 2025Peer-reviewedNeuroscience

Recapitulation and reversal of schizophrenia-related phenotypes in Setd1a-deficient mice

Jun Mukai, Enrico Cannavò, Gregg W. Crabtree, Ziyi Sun, Anastasia Diamantopoulou, Pratibha Thakur, Chia‐Yuan Chang, Yifei Cai, Stavros Lomvardas, Atsushi Takata, Bin Xu, Joseph A. Gogos

This study shows that Setd1a-deficient mice exhibit schizophrenia-like phenotypes, which can be reversed by restoring Setd1a expression or inhibiting LSD1.

Neuron · Oct 9, 2019Peer-reviewedNeuroscience