Author

Jonathan Rhys Carapetis

The Kids Research Institute AustraliaORCID 0000-0002-1182-9792Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1 Approach:statistical model· 1 Assay:whole-exome sequencing· 1

Frequent Co-Authors

Andrew C. SteeriD· 2 shared Alana L. WhitcombeiD· 1 shared Bo ReményiiD· 1 shared Ciara Baker· 1 shared Danika Lea HilliD· 1 shared Dawn C BessarabiD· 1 shared Genevieve SyniD· 1 shared Hannah R. C. FrostiD· 1 shared

Activity by Year

2017-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersImmunology

Andrew C. SteerORCID

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2 papers

Recent·Most cited

Streptococcus pyogenes pharyngitis elicits diverse antibody responses to key vaccine antigens influenced by the imprint of past infections.

Joshua Osowicki, Hannah R. C. Frost, Kristy I. Azzopardi, Alana L. Whitcombe, Reuben McGregor, Lauren H. Carlton, Ciara Baker, Loraine Fabri, Manisha Pandey, Michael F. Good, Jonathan Rhys Carapetis, Mark J. Walker, Pierre Robert Smeesters, Paul Vincent Licciardi, Nicole J. Moreland, , Andrew C. Steer

The paper examines antibody responses to S. pyogenes antigens in individuals with and without pharyngitis, finding differences in IgG and IgA levels and correlations with prior immunity.

Nature Communications · Dec 3, 2024Peer-reviewedImmunology

Genome-wide analysis of genetic risk factors for rheumatic heart disease in Aboriginal Australians provides support for pathogenic molecular mimicry

Lesley-Ann Gray, Heather A D’Antoine, Steven Y. C. Tong, Melita McKinnon, Dawn C Bessarab, Ngiare J Brown, Bo Reményi, Andrew C. Steer, Genevieve Syn, Jenefer Mary Blackwell, Michael Inouye, Jonathan Rhys Carapetis

This study identifies HLA-DQA1-DQB1 variants as genetic risk factors for rheumatic heart disease in Aboriginal Australians, supporting molecular mimicry as a pathogenic mechanism.

The Journal of Infectious Diseases · Sep 20, 2017Peer-reviewedGenetics