Jenna L. Cash

This study shows that persistent CCL3 expression in aged wounds sustains neutrophil recruitment and delays healing by impairing inflammatory resolution.

The paper identifies hypoxanthine as a key metabolite released during apoptosis that is consumed by gut bacteria, impairing epithelial repair.

This study develops a preclinical chronic wound model using snake venoms to characterize venom-induced wound pathophysiology and identify unique features.